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Here is a small sampling of Palmitoylethanolamide (PEA) studies by title:
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Palmitoylethanolamide (PEA) was identified in 1957. It is a naturally occurring compound made from fat and is produced by mammalian body's for the purpose of tissue repair. More specifically, PEA is an endogenous fatty acid amide formed, as the name implies, by combining a fatty acid with an amine (a family of organic compound containing nitrogen). The compound is one of the most common of the N-acylethanolamines (NAEs) [a family that includes anandamide (AEA) and oleoylethanolamide (OEA) a compound involved in feeling satieted].
PEA is not an endocannabinoid per se because it has no affinity (or only very weak affinity) for the classical endocannabinoid receptor sites CB1 or CB2. However, PEA may be an allosteric (indirect modulator) of CB1 and/or CB2.
PEA modulates other receptor sites that are part of the greater ECS-based environment entitled the endocannabinoidome (see the receptor site window for more information).
Foods that contain PEA include soy, egg yolk, corn, peanuts, pea and bean seeds. PEA was also discovered in human, bovine, and moose milk. Nutraceuticals containing PEA are readily available as oral supplements.
Palmitoylethanolamide, Palmidrol, 544-31-0, N-(2-Hydroxyethyl)hexadecanamide, Palmitoyl ethanolamide
IUPAC Name: N-(2-hydroxyethyl)hexadecanamide
Molecular Formula: C18H37NO2
Source–PubChem
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